In a client with Paget's disease, what primarily contributes to a stooped posture?

In Paget's disease, the primary contributor to a stooped posture is the process of bone resorption and regeneration. This condition leads to abnormal and excessive remodeling of bone tissue, resulting in weakened and deformed bones. As the disease progresses, the structural integrity of the affected bones, typically in the pelvis, skull, spine, and long bones, becomes compromised.

The imbalance between bone resorption and formation can result in enlarged and misshapen bones, and the spinal column may lose its normal curvature, leading to kyphosis (a hunchback appearance). This alteration in bone structure significantly affects posture, as the weakening of the vertebrae and changes in the spinal shape influence how the body aligns itself, often resulting in a stooped posture.

Muscle atrophy, joint inflammation, and weight-bearing strengthening are not the primary factors contributing to the stooped posture in this condition. While joint inflammation may occur, it does not directly lead to the postural changes seen in Paget's disease, and weight-bearing activities typically support bone density rather than contribute to stooped posture. Muscle atrophy might occur due to disuse but is not the main factor in this situation. Thus, the excessive bone resorption and aberrant regeneration primarily drive